会展名称 | 2012年载脂蛋白E阿兹海默症和脂蛋白生物学会议(AALB ) |
布展时间 | 2012年2月25日 |
展览时间 | 2012年2月26日—2012年3月2日 |
撤展时间 | 2012年3月4日 |
所在地址 |
美国 基斯通 |
会展场馆 | 基斯通度假胜地 |
参会注册费 | 可以. 会员490美元/非会员865美元 服务费300人民币/人 |
代送签证 | 可以. 服务费500人民币/人 |
代订机票 | 可以. 请与客服人员联系 |
代定展位 |
不可以 |
代定酒店 |
可以. 服务费为总费用15% |
会展描述 |
Apolipoprotein E4 (ApoE4) genotype is the primary risk factor for late-onset Alzheimer’s Disease. Approximately one fifth of the human population are ApoE4 carriers, yet more than 17 years after its discovery, the molecular basis by which this common allele causes neurodegeneration is still not understood. One major reason is the lack of effective communication between lipid researchers who have investigated the role of ApoE as a plasma lipid transport protein, and neuroscientists who are unfamiliar with the complex biochemistry of lipid metabolism and the cell biology of ApoE receptors. This is confounded by the methodological difficulty of studying lipid metabolism and lipoprotein transport in the brain and the paucity of pioneering work tackling this problem. Yet, the solution to one of the most pressing socioeconomic problem of our times, the looming Alzheimer’s Disease epidemic, depends on our ability to dissect and understand the molecular mechanism by which ApoE4 is causing this disease. The purpose of this highly interdisciplinary meeting on ApoE, Alzheimer’s and lipoprotein biology is to bring together a critical mass of experts with complementary expertise from these different fields, but who do not normally collaborate or attend the same conferences. In this manner, we envisage this meeting serving as the main catalyst for communication and collaboration between lipid and lipoprotein researchers, neurogeneticists, cell biologists and synaptic neurophysiologists. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on Clinical and Molecular Biology of Acute and Chronic Traumatic Encephalopathies, which will share opening keynote addresses and a fourth day of sessions with this meeting. 载脂蛋白E4(ApoE4)基因型是主要的风险因子迟发阿尔茨海默症。大约五分之一人口的人类ApoE4的载体,但超过17年后发现,这种分子基础原因是常见的等位基因仍然没有完全理解神经退化性。其中最主要的原因是缺乏有效的沟通了脂质研究人员的作用作为一个ApoE血脂转运蛋白,神经科学家,他们皆不熟悉复杂的生物化学对脂质代谢及细胞生物学的ApoE受体。这是蒙羞的研究方法论困难脂质代谢及脂蛋白转运的大脑与与外界隔绝。而解决这个问题的开创性工作。然而,解决一个最紧迫的社会经济问题的时候,阿尔茨海默病流行迫在眉睫,取决于我们的能力来解决和理解分子机制,ApoE4是导致这些疾病。本文的目的在高度跨学科会议ApoE、阿兹海默症和脂蛋白生物学是团结一个临界质量的专家有互补的专长,但从这些不同领域的人通常不合作或参加同一会议。以这样的方式,我们所设想的这次会议的主要催化剂作为沟通与合作,神经遗传学脂质和脂蛋白研究者、细胞生物学家和突触神经遗传学。跨学科的互动的机会将会明显的增强临床并发会议及其分子生物学的急性和慢性创伤神经遗传学开放基调,共享地址和第四天的会议在这次会议上。 |
展会周期 |
每年一届 |
注意事项 |
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