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 Acute traumatic brain injury (TBI) and its long-term neurodegenerative complications (known collectively as chronic traumatic encephalopathy, or CTE) are increasingly recognized as major public health issues, especially in the context of sports-related head injury and battlefield blast (military) exposure. Awareness of these issues has risen as neurological, psychiatric and neurodegenerative phenomena are associated with professional athletes and with veterans returning from Iraqi and Afghan theaters. The physics of head injury, as well as the epidemiological and neuropsychiatric aspects of sports-related and blast-related syndromes, will be reviewed in the initial sessions of this three-day meeting. Advances in functional neuroimaging and in the rapid and convenient determination of peptides and proteins in the cerebrospinal fluid and plasma will be reviewed in terms of their possible pathobiological significance as well as their identities as possible biomarkers. Later sessions will focus on the overlap between CTE and major neurodegenerative diseases, such as Alzheimer’s Disease (AD), frontotemporal dementia and amyotrophic lateral sclerosis. CTE is associated with accumulation of aggregated proteins or protein fragments, including tau, TDP-43, the Alzheimer’s amyloid precursor protein (APP) and the APP metabolite, amyloid-b. New experimental therapeutics research targets the roles these abnormal protein structures play in injury severity and subsequent outcome. Ab-lowering medications, currently in clinical trials for AD, improve outcome following acute brain trauma in rodent models, suggesting a pathway toward potential therapeutic interventions. Because apolipoprotein E (APOE) isotype is an important determinant of outcome from acute severe as well as mild repetitive TBI, this meeting coincides in time and venue with the Keystone Symposia meeting on ApoE, Alzheimer’s and Lipoprotein Biology. Opening plenary sessions and an optional fourth day of sessions are available for those interested in learning more about basic biology and/or clinical interventions related to APOE.

急性创伤性脑损伤(TBI)以及其长期退行性并发症(合称为慢性创伤性脑病、或查询语句太复杂)已经被越来越多的人认为主要的公共卫生问题,特别是在运动背景下的头部受伤(军事)及战场爆炸曝光。意识到这些问题已经上升为精神疾病和神经退化现象的神经系统,相关的职业运动员,从伊拉克和阿富汗退伍军人返回上映。头部受伤的物理特性,以及流行病学和精神方面的信息和相关联证候,治疗方法进行了回顾在初始届这个连续三天的会议。先进的功能成像和方便快捷的方法测定多肽和蛋白质在脑脊液和等离子体进行了回顾从其可能的pday意义以及他们的身分作为可能的生物标记物。后来会议将集中重叠的查询语句太复杂和主要神经退行性疾病,如阿尔茨海默病(AD)、痴呆、额颞肌萎缩性脊髓侧索硬化症。有关的查询语句太复杂聚集蛋白积累或蛋白质碎片,包括陶、TDP-43,阿尔茨海默氏病淀粉样前体蛋白(APP)和应用程序,含淀粉代谢物。新的实验疗法的研究对象中所扮演的角色扮演这些不正常的蛋白质结构在损伤严重度和随后的结果。劣质药物,目前在临床试验结果为广告,改善急性脑外伤后在动物模型中,提出一个潜在的治疗方法途径。因为载脂蛋白E(APOE)是一个重要的决定因素同样结果急性严重脑外伤以及轻微的重复,这会在时间和地点一致的学术会议上重点APOE、阿兹海默症和脂蛋白生物学。开放全体会议和一个可选的第四天的课程供那些有兴趣去学习更多有关基本生物学和/或临床干预措施与APOE。

 每年一届　

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 2.单项服务的费用以最终确认单为准.
 3.参会整体流程服务费1000人民币/人.
   服务费包含:代送签证、代订往返机票、代订当地酒店.  
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