可以. 服务费为总费用15%

 Cellular functions depend on numerous protein-coding and noncoding RNAs and the RNA-binding proteins (RBPs) associated with them, which form ribonucleoprotein complexes (RNPs). Mutations that disrupt either the RNA or protein components of RNPs or the factors required for their assembly can be deleterious. The discovery of disease-causing mutations in RNAs or their interacting RBPs not only adds to our growing understanding of RNA biology and chemistry, but also offers new therapeutic targets and contributes to ongoing efforts in developing mRNA-targeting antisense and RNAi therapeutics. This meeting focuses on a general understanding of post-transcriptional gene regulation and how perturbation of complex networks of RNA-protein and RNA-RNP interactions triggers disease.
 
细胞功能依赖于许多蛋白质编码和非编码RNA的RNA结合蛋白（限制性商业惯例）与之关联，形成核蛋白复合物（RNPs）。可以是有害的基因突变，破坏RNA或蛋白质成分或RNPs其组装所需的因素。发现致病突变的RNA或相互作用的限制性商业惯例，不仅增加了我们不断增长的RNA生物学和化学的理解，但也提供了新的治疗靶点，促进发展mRNA的定位反义和RNAi疗法的持续努力。本次会议的重点在转录后的基因表达调控和的RNA -蛋白质和RNA- RNP相互作用的复杂网络的扰动触发疾病的一般理解。

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