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 Diabetes is associated with significantly accelerated rates of several microvascular and macrovascular complications including nephropathy, neuropathy, retinopathy and atherosclerosis. Furthermore, results from both clinical trials and animal studies indicate that these complications might progress despite glycemic control, a phenomenon termed metabolic memory. Ongoing studies have identified biochemical and nuclear mechanisms, genetic determinants, epigenetic factors and non-coding RNAs in the pathogenesis of diabetic complications. Thus, the study of the key mechanisms leading to injury in target organs and failure of adaptive processes holds great promise for the identification of mechanism-targeted therapeutic approaches. These experimental approaches have been aided by tremendous advances in high-throughput genomic and epigenomic technologies, integrated systems biology and bioinformatics. Advances in the fields of stem cell therapies, induced pluripotency and regenerative medicine have provided new hope for the treatment of complications. The goal of this interdisciplinary meeting is to integrate these evolving concepts with current knowledge to facilitate a systems-level understanding of diabetic complications.

糖尿病是与几个微血管和大血管并发症，包括肾病，神经病变，视网膜病变和动脉粥样硬化率明显加快。此外，从临床试验和动物实验研究结果表明，这些并发症的可能，尽管血糖控制方面取得进展，这种现象被称为代谢记忆体。正在进行的研究已经确定了生化和核机制，遗传因素，表观遗传因素在糖尿病并发症的发病机制中的非编码RNA。因此，导致靶器官损伤和适应过程失败的主要机制的研究认为大有希望的识别机制，有针对性的治疗方法。这些实验方法已资助在高通量的基因组和表观基因技术，集成系统生物学和生物信息学的巨大进步。在诱导多能性和再生医学，干细胞疗法领域的进展，有并发症的治疗提供了新的希望。这个跨学科的会议的目标是整合现有的知识，这些不断发展的的概念，以方便了解系统级的糖尿病并发症。

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