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 Although altered glucose metabolism was first noted as a characteristic of tumor cells by Otto Warburg in the 1920s, the molecular underpinning for this observation did not begin to be appreciated fully until the past decade. This area has exploded over the last five years with the identification of extensive interactions of genuine tumor suppressor genes and oncogenes with metabolic control. The increased biosynthetic needs of tumor cells demand a reprogramming of cellular metabolism, which also creates increased energy demands and makes tumor cells more vulnerable to interventions targeting their metabolism. The goal of the Keystone Symposia meeting on Cancer and Metabolism is to bring together leaders in the fields of cancer and metabolic control with those studying the signaling pathways linking and governing each. The latest discoveries will be discussed as well as how this knowledge can be exploited to design future cancer therapeutics. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on Advances in Hypoxic Signaling: From Bench to Bedside, which will share a keynote address and a plenary session with this meeting.

虽然葡萄糖代谢改变是第一家Otto Warburg特征的肿瘤细胞，在20世纪20年代所指出的，这种观察的分子基础，没有开始要充分赞赏，直到过去十年。这一地区在过去的五年爆炸识别真正的肿瘤抑制基因和癌基因与代谢控制的广泛的互动。增加了肿瘤细胞的生物合成需要的需求，重新编程细胞的新陈代谢，这也造成了能源需求增加，使肿瘤细胞更容易受到针对其代谢。癌症和代谢梯形的专题讨论会会议的目的是汇集癌症和代谢控制研究的信号通路，连接和控制每个领域的领导者。以及如何可以利用这方面的知识，来设计未来的的癌症治疗的最新发现，将讨论。跨学科的互动的机会将大大增强并发会议上的缺氧信号的进展：从实验室到床边，将与本次会议的主题演讲和全体会议。

 每年一届　

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