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 Post-translational modifications of cellular proteins by ubiquitin and ubiquitin-like modifiers regulate a variety of functions in the cell. A crucial aspect of signaling through ubiquitin and Ubls is the presence of modular binding domains that recognize particular types of ubiquitin signals. Such molecular coupling creates an attractive “decoding” mechanism for responding to alterations in the cellular microenvironment and regulating physiological and pathophysiological conditions. In this meeting, we will discuss the major challenges in the field including detection of endogenous ubiquitination events, functional aspects of novel types of ubiquitin chains and their recognition by selective binding domains, and high-throughput technologies for comprehensive analysis of Ub modifications. The intersection between the deregulated ubiquitin signaling, pathogenesis of human diseases and therapies targeting the Ub system will be explored. The program offers a possibility for young scientists to present their own work and engage in discussions on the emerging topics in the field. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on NF-kB Signaling and Biology: From Bench to Bedside, which will share a keynote address and three plenary sessions with this meeting.

细胞蛋白翻译后修饰泛素和泛素样修饰符的调节多种细胞的功能。通过泛素和Ubls信号的一个关键方面是模块化结合结构域识别特定类型的泛素信号的存在。这种分子耦合创建一个有吸引力的的“解码”机制，在细胞微环境的改变和调节生理和病理条件下。在这次会议上，我们将讨论在该领域的重大挑战，包括泛素内源性事件，选择性结合域的泛素链和他们的认同的小说类型的功能性方面，和综合分析泛修改的高通量技术的检测。放松管制的泛素信号之间的交叉，将探讨人类疾病的发病机理和治疗方法，针对美国布法罗大学的系统。该方案提供了可能性，青年科学家目前自己的工作和从事在该领域的新兴议题的讨论。跨学科的互动机会将显着增强，NF - kB信号和生物并发会议：从实验室到床边，将与本次会议上发表专题演说和三次全体会议。

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