可以. 服务费为总费用15%

 Mounting evidence indicates that cell death in diseases results from complex interactions between molecular mechanisms taking place within the degenerating cells (cell-autonomous) and outside the degenerating cells (non-cell autonomous). At the cross roads of these diverse signaling pathways are found the mitochondria which, through proper mitochondrial dynamics and regulated mitochondrial elimination by autophagy (i.e., mitophagy), play a crucial role in maintaining the well-being of the cells. This meeting will focus on the general mechanisms of cell death including the Bcl-2 family and caspase proteases, the role of mitochondrial dynamics and mitophagy, the usefulness of bioinformatics to unravel death-pathways, the contribution of non-cell autonomous mechanisms to cell death and the therapeutic strategies to protect against cell death. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on Mitochondrial Dynamics and Function, which will share a keynote address and two plenary sessions with this meeting.

越来越多的证据表明，细胞死亡的疾病从内退化的细胞（细胞自治）和外变质细胞（非细胞自主）发生的分子机制之间复杂的相互作用的结果。在这些不同的信号转导通路的交叉道路被发现的线粒体，通过适当的线粒体动态和自体吞噬，受线粒体消除，发挥了至关重要的作用，在维持细胞的福祉。本次会议将重点放在一般包括BCL -2家庭和细胞凋亡蛋白酶，作用线粒体动力学，生物信息学的实用性，揭开死亡的途径，非细胞自治机制的贡献细胞死亡细胞死亡的机制和治疗策略，以防止细胞死亡。跨学科的互动的机会将显着增强线粒体的动态和功能，这将与本次会议上发表主题演讲和两次全体会议并发会议。

 每年一届　

 1.以上标注"可以"的服务项目可单独办理.
 2.单项服务的费用以最终确认单为准.
 3.参会整体流程服务费1000人民币/人.
   服务费包含:代送签证、代订往返机票、代订当地酒店.  
   服务费不含:各项目所产生的实际费用.

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