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 Mitochondria are no longer considered “only” as the cellular thermodynamic converter: mounting evidence implicates them as key organelles in cell signaling, proliferation, aging, disease and death. Indeed, our view of this organelle was profoundly altered with the discovery that mitochondria function within a connected network that is continuously remodeled by fusion and division events. This reality has sparked and accelerated new scientific discoveries through the intersection of different fields about the functional roles and molecular features of mitochondrial behaviors. The interaction between the cell death and mitochondrial dynamics fields is the best example of such interplay and has led to the discovery that modulators governing cell death and mitochondrial dynamics, respectively, are in fact interacting with one another. This Keystone Symposia meeting uniquely builds on this rich interdisciplinary interaction, which continues to provide novel insights into the mechanisms underlying mitochondrial behavior and mitochondrial-associated diseases. Opportunities for interdisciplinary interactions will be significantly enhanced by the concurrent meeting on Cell Death Pathways: Beyond Apoptosis, which will share a keynote address and two plenary sessions with this meeting.

线粒体不再被认为是“唯一”作为蜂窝热力学转换器：越来越多的证据牵连他们在细胞信号转导，细胞增殖，衰老，疾病和死亡的重要细胞器。事实上，我们深刻地改变这种细胞器与发现线粒体的功能，在连接的网络融合和分裂活动，不断改造。这一现实已经引起并加速通过线粒体行为的职能作用和分子特征的不同领域的交叉科学新发现。细胞死亡和线粒体动力学领域之间的相互作用，这种相互作用的最好的例子，并导致发现，调制器，细胞死亡和线粒体动态，分别在彼此互动。这梯形研讨会上唯一建立在这个丰富的跨学科的互动，进入线粒体的行为和线粒体相关疾病的基本机制，继续提供新的见解。跨学科的互动机会将显着增强细胞死亡途径并发会议：除了细胞凋亡，将与本次会议上发表主题演讲和两次全体会议。

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